Syndromes of peripheral motor neuron insufficiency and autonomic maladjustment in patients with infantile cerebral palsy in the late residual period

Following perinatal brain lesions, disneuroontogenetic phenomena in infantile cerebral palsy (CP) cause the formation of a multisystem neurological pathology in the residual period of this disease, which manifests itself in complex, often specific, clinical phenomena that distinguish the neurological chemotics of cerebral palsy. As clinical experience shows, along with well-known central disorders of the animal nervous system, in the general syndromology of cerebral palsy of the late period, a certain place can be occupied by secondary, disneuroontogenetically determined lesions of the peripheral and autonomic nervous systems.